讓自己更好的生活提案論文書籍站
臺北醫學大學 醫學科學研究所博士班 林良宗、RICHARDSON, CHRISTOPHER所指導 劉卿宣的 開發Nectin-4標靶之溶瘤麻疹病毒療法作為乳癌及肝癌治療 （2020），提出iNO S7關鍵因素是什麼，來自於Measles virus、Oncolytic virotherapy、Breast cancer、Liver cancer、Camptothecin、Ursolic acid、Nanoparticles、Chemovirotherapeutic、Nectin-4。
而第二篇論文國立臺灣師範大學 物理學系 劉祥麟所指導 葉秦維的 摻雜鑭系元素(鏑,釓)氧化鋅與單層二(硫,硒)化鎢薄膜的光譜性質研究 （2014），提出因為有 氧化鋅、過渡金屬二硫屬化物、拉曼散射光譜、橢圓偏振光譜、光譜性質的重點而找出了 iNO S7的解答。
iNO S7進入發燒排行的影片
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👉https://amy77.com/blog/post/33627935
開發Nectin-4標靶之溶瘤麻疹病毒療法作為乳癌及肝癌治療
為了解決iNO S7 的問題，作者劉卿宣 這樣論述：
Oncolytic viruses (OVs) are a rapidly emerging treatment platform for various types of cancer that offers several advantages over conventional anticancer therapeutics, including selectivity, potency, and targeted cell death. Recent studies have revealed that measles virus (MV), a member of the Para
myxoviridae family, possesses the ability to selectively target carcinomas by recognizing the tumor cell marker nectin-4, which is highly expressed in many types of cancer. Specifically, wild-type MV targets nectin-4 on the tumor cells and does not engage CD46, which is an additional receptor expres
sed on all nucleated cells and employed by vaccine strains of MV for infection. The use of MV-based vectors with wild-type glycoproteins is thus justified as candidate for potential development as an oncolytic agent targeting nectin-4. The results in this thesis present as proof-of-concept, the use
of a recombinant wild-type MV as an oncolytic agent to effectively target nectin-4-marked cancers, and demonstrate its ability to induce oncolysis of nectin-4-expressing breast and hepatocellular carcinoma (HCC) tumor cells. Specifically, it was observed that MV could dose-dependently and efficientl
y target and induce cytotoxicity in both cancer models. In addition, the data also demonstrate the feasibility to use a combination of chemotherapeutic agents or anticancer nanoparticles as a chemovirotherapeutic approach to enhance MV’s oncolytic efficacy, while reducing the doses of treatment requ
ired and potentially increasing its safety. Results from this study underpin the oncolytic potential of recombinant MV and support the future development of oncolytic virotherapy using MV-based vectors for the management of nectin-4-expressing cancers.
摻雜鑭系元素(鏑,釓)氧化鋅與單層二(硫,硒)化鎢薄膜的光譜性質研究
為了解決iNO S7 的問題，作者葉秦維 這樣論述：
我們量測摻雜（鏑,釓）氧化鋅薄膜的拉曼散射光譜、穿透光譜及橢圓偏振光譜，進而研究不同摻雜量對氧化鋅薄膜光譜性質的影響。此外，我們量測單層過渡金屬二硫屬化物薄膜（二硫化鎢和二硒化鎢）的拉曼散射光譜及橢圓偏振光譜，進而探討單層二硫化鎢和二硒化鎢薄膜的光譜性質。氧化鋅薄膜是用脈衝雷射沉積法製成在藍寶石基板上，摻雜鏑離子和釓離子的濃度範圍分別為1% ~ 10%及3% ~ 30%。單層二硫化鎢和二硒化鎢薄膜是用化學氣相沉積法製成在藍寶石基板上。這篇論文的目的是探討上述所有材料的晶格結構和電子結構。我們發現純氧化鋅薄膜的拉曼散射光譜，顯示2個拉曼峰，其頻率位置為98.7 cm-1和437.1 cm-1,
分別為E2(low)和E2(high)對稱性。隨著鏑離子和釓離子摻雜濃度增加，拉曼峰E2(low)和E2(high)的峰值強度會逐漸下降。在穿透光譜中發現，隨著鏑離子和釓離子摻雜濃度增加，（鏑,釓）氧化鋅薄膜在紫外光區的光穿透率會提高。在吸收能譜中發現，隨著鏑離子和釓離子摻雜濃度增加，氧化鋅薄膜的直接能隙值會受到鏑離子和釓離子的影響，產生偏移，其現象可被能帶隙變窄理論和伯斯坦-莫斯位移理論解釋。我們發現單層二硫化鎢和二硒化鎢薄膜在532奈米雷射光激發下的拉曼散射光譜具有多種類的拉曼峰。在室溫的吸收能譜中發現，單層二硫化鎢和二硒化鎢薄膜具有明顯的激子A和B吸收峰。此外，我們分析了單層二硫化鎢和二
硒化鎢薄膜的室溫直接能隙值和激子束縛能值。其室溫直接能隙值，分別為2.1電子伏特和1.72電子伏特；其室溫激子束縛能值，分別為0.32電子伏特和0.24電子伏特。在變溫的吸收能譜中發現，單層二硫化鎢和二硒化鎢薄膜的直接能隙值會產生紅移現象，此現象是由單層二硫化鎢和二硒化鎢薄膜的晶格受到熱膨脹和電子及聲子間的交互作用所造成。